1. Atcheson, S.G., Ward, J.R., Lowe, W., “Concurrent medical disease in work-related carpal tunnel syndrome.” Arch Intern Med, 1998; 158:1506-1512.
Concludes CTS tends to occur in people with lost of other physical problems. Therefore, whether work related or decline in general health becomes the unanswered question.
2. Baba, M., Ozaki, I., Watahiki, Y., Kudo, M., Takebe, K., and Matsunaga, M. “Focal conduction delay at the carpal tunnel and the cubital fossa in diabetic polyneuropathy.” Neurophysiol, 1987; 27:119-123.
Confirms that even with diabetic neuropathy localized entrapment can be superimposed. Ties in with the work of A.L. Dellon.
3. Bonatz, E., and Seabol, K.E., CHT. “Acute carpal tunnel syndrome in a patient taking Coumadin: Case report.” Journal of Trauma, 1993; 35:1: 143-144.
Example of how increasing the contents by bleeding in the carpal tunnel may increase pressure.
4. Bradish, C.F. “Carpal tunnel syndrome in patients on haemodialysis.” JBJS, 1985; 67B: 1: 130-132.
Demonstrates how fluid retention from kidney failure may be implicated in some cases of CTS and how CTR solves the symptoms.
5. Chamma, M., Bousquet, P., Renard, E., “Dupuytren’s disease, carpal tunnel syndrome, trigger finger and diabetes mellitus.” JHS, 1995; 20A: 109-14.
Another article revealing high frequency of multiple problems in the same person. The disease related only by genetics.
6. Florack, T.M., Gastonia, NC, Miller, R.J., Pellegrini, V.D., Buront, R.I., Dunn, M.G. Rochester, NY. “The prevalence of carpal tunnel syndrome in patients with basal joint arthritis of the thumb.” JHS 1992; 17A: 624-30.
Review of literature and study of their own patients reveals greater than 50% incidence of CTS in association with basilar thumb arthritis.
7. Gamstedt, A., Holm-Glad, J., Ohlson, C.G., Sundstrom, M., “Hand abnormalities are strongly associated with the duration of diabetes mellitus.” J of Intern Med, 1993; 234:189-193.
Ties into the well documented increase in all sorts of systemic pathologies in long standing diabetics.
8. Kleinman, W.B, Bishop, A.T., “Anterior intramuscular transposition of the ulnar nerve.” JHS, 1989; 14A: 972-979.
Study of patients undergoing cubital tunnel decompression reveals high association with CTS of ~60%.
9. Knezevic, W., Masstaglia, F.L., “Neuropathy associated with Brescia-Cimino arteriovenous fistulas.” Arch Neurol, 1984; 41: 1184-1186.
Ties into studies on ischemia (cutting off circulation).
10. Kremchec, T.E., Kremchek, E.J., “Carpal tunnel syndrome caused by flexor tendon sheath lipoma.” Ortho Review, 1988; XVII: 11.
Another flawed case report. Whether or not the lipoma was causally related to CTS development is speculative without pressure measurement.
11. Murray-Leslie, C.F., Wright, V., “Carpal tunnel syndrome, humeral epicondylitis, and the cervical spine: a study of clinical and dimensional relations.” BMJ, 1976; 1: 1439-1442.
This was a retrospective look at 43 patients treated surgically for carpal tunnel syndrome and comparing them with 43 controls noting the high prevalence of lateral epicondylitis (33%) in the CTS patients vs. 7% in the control. Blind reading of the cervical spine x-rays on all of the subjects reveal a consistent trend to degenerative cervical spine disease in those people treated for carpal tunnel syndrome than in the control. The lesions were significant in several vertebral levels. Additionally, they had medial humeral epicondylitis in 3 patients (this is usually caused by cubital tunnel syndrome). I also wanted to note that the carpal tunnel group did not differ in body height or weight from the control group. They recorded the possibility that narrowing of the cervical disc might be evidence of degeneration of soft tissues diffusely about throughout the body, such as a common extensor origin at the elbow resulting in epicondylalgia.
12. Nakamichi, K., Tachibana, S., Tokyo, Japan, Tamai, K., Tochigi, Japan. “Carpal tunnel syndrome caused by a mass of calcium phosphate.” JHS, 1994; 19A: 1: 111-113.
Speculative cause and effect case report. No value.
13. O’Duffy, J.D., Randall, R.V., MacCarty, C.S. Rochester Minn. “Median Neuropathy (Carpal tunnel syndrome) in acromegaly.” Ann Int Med, 1973; 78:379-383.
Strongly suggests CTS is acromegaly (gigantism) resolved with hormone correction. No hand science and no long term follow up.
14. Robbins, H., New York, NY. “Anatomical study of the median nerve in the carpal tunnel and etiologies of the carpal tunnel syndrome.” Hospital of J Diseases, 1963; 45A: 5.
A simple review of the shape, size, and inter relationship of anatomic structures about the carpal canal.
15. Voitk, A.J., Mueller, J.C., Farlinger, D.E., Johnston, R.U., “Carpal tunnel syndrome in pregnancy.” Can Med Assoc J, 1983; 128.
High incidence of CTS symptoms in pregnant women of ~80% leaving only a few symptomatic after child delivery. No long term follow up, however, to see how these same women faired years later.
16. Windelmann, R.K., Connolly, S.M., and Doyle, J.A., Rochester MN. “Carpal tunnel syndrome in cutaneous connective tissue disease: Generalized morphea, lichen sclerosis, fascitis, discoid lupus erythematosus, and lupus erythematosus, and lupus panniculitis.” J Am Acad Dermatol, 1982; 7:94-99.
Without NCS or CT pressure data causal relationship between CTS and these diseases is speculative.
17. Yu, J., Bendler, E.M., Mentari, A. “ Neurological disorders associated with carpal tunnel syndrome.” Electromyogr Clin Neurophysiol, 1979; 19, 27-32.